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Dr. Kerry Tarvin

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Spinal Decompression

Why Spinal Disc Decompression Works:

This FDA cleared technology relieves pain by enlarging the space between the discs. The negative pressure of decompression releases pressure that builds on to the disc and nerves, allowing the herniated and bulging disc to eventually go back into normal position. Decompression is the only treatment that is truly most effective for severe cases of herniation, degeneration, arthritis, stenosis and pressure on the nerve root. According to a clinical study performed by the Orthopedic Technological Review in 2004, said that 86% of all cases experienced spinal pain relief with disc decompression.

What is a  Disc? What does a Disc do?

The disc is a soft cartilaginous material that seperates the spinal vertabrae. We have Discs starting at the the third cervical  all the way down to the lowest Lumbar  vertabrae L 5. The Discs middle is called the nucleus and and outer layer called the annulus.These discs act as shock absorbers and these disc after we are done growing usually arounf 19 years old become avascualr (does not receive a good blood supply).  One of the reasons that our discs give us so many problems is that it does not heal well without a good blood supply. When we break a bone it heals quickly and actually stronger than before! Our discs are avascular so if a disc is injured it continues to degenerate and does not heal properly. One of the amazing benefits of decompression is that it heightens the disc space, and is seen to return it to normal size and function.  

The Decompression therapy also causes the disc to heal because the treatment stimulates the cartildge to regenerate and act it cannot do on its own without the decompression treatment.Spinal Decompression also causes the nutrient rich blood to fill the disc space which fosters the healing response.

Dr. Kerry Tarvin DC DABCO will take a complete  initial history, perform a physical evaluation, review all data including MRI´s  to  prequalify  the patient to ensure the decompression procedures will benefit the patient.
   Dr Tarvin has been expertly trained in the delivery of  spinal decompression and has  dedicated a large portion of his practice to the relief of severe and chronic low back pain and cervical pain and assisting the patient to return to a normal, pain free life.

Bulging Disc

To really understand what a Bulging disc I will discuss the classifications to describe the disc lesions because there is a plethora of terms commonly encountered like  Disc Prolapse, Slipped Disc,ruptured disc,disk herniation.

The term Disc Herniation implies that a rupture or a tear of the annular fibers occurs allowing the migration of nucleur material beyond the verterbral margin. The nuclear material may protrude out and cause a distention of the outer annulus fibrosis or rupture through the annulus and extrude behind the posterior ligament.

Four classifications exist to describe disc lesions

1. Annular bulge

2. Protrusion (Herniation)

3. Extrusion

4. Free Disc Fragment (Sequestration).

The annular bulge or Disc bulge is a small disc herniation that does not directly contact the nerve root and left untreated can easily progress to a larger degree of nuclear protrusion because of the loss of the annular fibers to contain the nucleus. Degeneration will predispose annular fibers to failure following trauma.

Disc Protrusion (Herniation) A Disc herniation represents a rupture of nuclear material through a defect in  annulus, producing a focal extension of  the disc or a broad based extension of the disc margin.Intervertebral disc herniations result in some degree from central canal or foraminal occlusion.

Disk Extrusion applies when a portion of the nucleus pulposus fibrocartilage and end plate cartilage have migrated through comprised outer annular fibers. Disc extrusions may compress the root, cord or both and can cause myeloradiculopathy( shooting pain).

Free disc Fragment (Sequestered Disc) refers to the extra-annular seperation and migration of a piece of nuclear material. Cauda Equina syndrome are common with intradural migration. This is when you lose control of bowel and bladder function. This is a emergency situatiion and usually requires surgery to correct the problem.

The most common area is the Lower Lumbar vertabre L5. The pressure causes the jelly material inside the disc to "bulge" or "slip" out of place .The bulge itself puts pressure on  the nerves( usually the spinal nerve).The Spinal nerve is very sensitive even a small amount of pressure causes the nerve to dysfunction. The most common cervical disc to herniate is also the fifth  and can cause cervicobrachial syndrome or symptoms like carpal tunnel sydrome or weakness in the arms or a burning sensation in the arms.

Disc Dessication is when the annullar fibers degenerate due to dehydration, One preventative step is to drink as much water as can if you feel that you have or are developing a Disc Herniation.

Leg Pain or Sciatica comes from bulging discs pressing on spinal nerves or spinal cord.

Pain in legs like Sciaitica

The Sciatic nerve is the largest nerve in the body and it is a common problem with people and when a disc herniates it can causes Sciaitica which can feel like a sharp shooting pain down the look into the buttocks or even into the toes. In extreme examples it can cause permanent nubmness and loss of muscle strength.

  Decompression technology

NASA does not endorse any equipment or table nor do they claim that this was their discovery.

. The FDA cleared  several tables to be caled Spinal Disc decompression and they all do basically the same thing some have fancier options. Some of these tables have names like the spine med, the lordex machine, ABS, Extentrac, 3D active trac.

Spinal Decompression causes  the Disc material to get sucked back into the  joint space and the outer layer is made stronger  by stimulating the cartilidge  to regenerate to prevent re-injury.

Spinal Decompression

By Thomas A. Gionis, MD, JD, MBA, MHA, FICS, FRCS, and Eric Groteke, DC, CCIC

The outcome of a clinical study evaluating the effect of nonsurgical intervention on symptoms of spine patients with herniated and degenerative disc disease is presented.

This clinical outcomes study was performed to evaluate the effect of spinal decompression on symptoms and physical findings of patients with herniated and degenerative disc disease. Results showed that 86% of the 219 patients who completed the therapy reported immediate resolution of symptoms, while 84% remained pain-free 90 days post-treatment. Physical examination findings showed improvement in 92% of the 219 patients, and remained intact in 89% of these patients 90 days after treatment. This study shows that disc disease, the most common cause of back pain, which costs the American health care system more than $50 billion annually can be cost-effectively treated using spinal decompression. The cost for successful non-surgical therapy is less than a tenth of that for surgery. These results show that biotechnological advances of spinal decompression reveal promising results for the future of effective management of patients with disc herniation and degenerative disc diseases. Long-term outcome studies are needed to determine if non-surgical treatment prevents later surgery, or merely delays it.

INTRODUCTION: ADVANCES IN BIOTECHNOLOGY

With the recent advances in biotechnology, spinal decompression has evolved into a cost-effective nonsurgical treatment for herniated and degenerative spinal disc disease, one of the major causes of back pain. This nonsurgical treatment for herniated and degenerative spinal disc disease works on the affected spinal segment by significantly reducing intradiscal pressures.1 Chronic low back pain disability is the most expensive benign condition that is medically treated in industrial countries. It is also the number one cause of disability in persons under age 45. After 45, it is the third leading cause of disability.2 Disc disease costs the health care system more than $50 billion a year.

The intervertebral disc is made up of sheets of fibers that form a fibrocartilaginous structure, which encapsulates the inner mucopolysaccharide gel nucleus. The outer wall and gel act hydrodynamically. The intrinsic pressure of the fluid within the semirigid enclosed outer wall allows hydrodynamic activity, making the intervertebral disc a mechanical structure.3 As a person utilizes various normal ranges of motion, spinal discs deform as a result of pressure changes within the disc.4 The disc deforms, causing nuclear migration and elongation of annular fibers. Osteophytes develop along the junction of vertebral bodies and discs, causing a disease known as spondylosis. This disc narrows from the alteration of the nucleus pulposus, which changes from a gelatinous consistency to a more fibrous nature as the aging process continues. The disc space thins with sclerosis of the cartilaginous end plates and new bone formation around the periphery of the contiguous vertebral surfaces. The altered mechanics place stress on the posterior diarthrodial joints, causing them to lose their normal nuclear fulcrum for movement. With the loss of disc space, the plane of articulation of the facet surface is no longer congruous. This stress results in degenerative arthritis of the articular surfaces.5

This is especially important in occupational repetitive injuries, which make up a majority of work-related injuries. When disc degeneration occurs, the layers of the annulus can separate in places and form circumferential tears. Several of these circumferential tears may unite and result in a radial tear where the material may herniate to produce disc herniation or prolapse. Even though a disc herniation may not occur, the annulus produces weakening, circumferential bulging, and loss of intervertebral disc height. As a result, discograms at this stage usually reveal reduced interdiscal pressure.

The early changes that have been identified in the nucleus pulposus and annulus fibrosis are probably biomechanical and relate to aging. Any additional trauma on these changes can speed up the process of degeneration. When there is a discogenic injury, physical displacement occurs, as well as tissue edema and muscle spasm, which increase the intradiscal pressures and restrict fluid migration.6 Additionally, compression injuries causing an endplate fracture can predispose the disc to degeneration in the future.

The alteration of normal kinetics is the most prevalent cause of lower back pain and disc disruption and thus it is vital to maintain homeostasis in and around the spinal disc; Yong-Hing and Kirkaldy-Willis7 have correlated this degeneration to clinical symptoms. The three clinical stages of spinal degeneration include:

1. Stage of Dysfunction. There is little pathology and symptoms are subtle or absent. The diagnosis of Lumbalgia and rotatory strain are commonly used.

2. Stage of Instability. Abnormal movement of the motion segment of instability exists and the patient complains of moderate symptoms with objective findings. Conservative care is used and sometimes surgery is indicated.

3. Stage of Stabilization. The third phase where there are severe degenerative changes of the disc and facets reduce motion with likely stenosis.

Spinal decompression has been shown to decompress the disc space, and in the clinical picture of low back pain is distinguishable from conventional spinal traction.8,9 According to the literature, traditional traction has proven to be less effective and biomechanically inadequate to produce optimal therapeutic results.8-11 In fact, one study by Mangion et al concluded that any benefit derived from continuous traction devices was due to enforced immobilization rather than actual traction.10 In another study, Weber compared patients treated with traction to a control group that had simulated traction and demonstrated no significant differences.11 Research confirms that traditional traction does not produce spinal decompression. Instead, decompression, that is, unloading due to distraction and positioning of the intervertebral discs and facet joints of the lumbar spine, has been proven an effective treatment for herniated and degenerative disc disease, by producing and sustaining negative intradiscal pressure in the disc space. In agreement with Nachemon´s findings and Yong-Hing and Kirkaldy-Willis,1 spinal decompression treatment for low back pain intervenes in the natural history of spinal degeneration.7,12 Matthews13 used epidurography to study patients thought to have lumbar disc protrusion. With applied forces of 120 pounds x 20 minutes, he was able to demonstrate that the contrast material was drawn into the disc spaces by osmotic changes. Goldfish14 speculates that the degenerated disc may benefit by lowering intradiscal pressure, affecting the nutritional state of the nucleus pulposus. Ramos and Martin8 showed by precisely directed distraction forces, intradiscal pressure could dramatically drop into a negative range. A study by Onel et al15 reported the positive effects of distraction on the disc with contour changes by computed tomography imaging. High intradiscal pressures associated with both herniated and degenerated discs interfere with the restoration of homeostasis and repair of injured tissue.

Biotechnological advances have fostered the design of Food and Drug Administration-approved ergonomic devices that decompress the intervertebral discs. The biomechanics of these decompression/reduction machines work by decompression at the specific disc level that is diagnosed from finding on a comprehensive physical examination and the appropriate diagnostic imaging studies. The angle of decompression to the affected level causes a negative pressure intradiscally that creates an osmotic pressure gradient for nutrients, water, and blood to flow into the degenerated and/or herniated disc thereby allowing the phases of healing to take place.

This clinical outcomes study, which was performed to evaluate the effect of spinal decompression on symptoms of patients with herniated and degenerative disc disease, showed that 86% of the 219 patients who completed therapy reported immediate resolution of symptoms, and 84% of those remained pain-free 90 days post-treatment. Physical examination findings revealed improvement in 92% of the 219 patients who completed the therapy.

METHODS

The study group included 229 people, randomly chosen from 500 patients who had symptoms associated with herniated and degenerative disc disease that had been ongoing for at least 4 weeks. Inclusion criteria included pain due to herniated and bulging lumbar discs that is more than 4 weeks old, or persistent pain from degenerated discs not responding to 4 weeks of conservative therapy. All patients had to be available for 4 weeks of treatment protocol, be at least 18 years of age, and have an MRI within 6 months. Those patients who had previous back surgery were excluded. Of note, 73 of the patients had experienced one to three epidural injections prior to this episode of back pain and 22 of those patients had epidurals for their current condition. Measurements were taken before the treatments began and again at week two, four, six, and 90 days post treatment. At each testing point a questionnaire and physical examination were performed without prior documentation present in order to avoid bias. Testing included the Oswetry questionnaire, which was utilized to quantify information related to measurement of symptoms and functional status. Ten categories of questions about everyday activities were asked prior to the first session and again after treatment and 30 days following the last treatment.

Testing also consisted of a modified physical examination, including evaluation of reflexes (normal, sluggish, or absent), gait evaluation, the presence of kyphosis, and a straight leg raising test (radiating pain into the lower back and leg was categorized when raising the leg over 30 degrees or less is considered positive, but if pain remained isolated in the lower back, it was considered negative). Lumbar range of motion was measured with an ergonometer. Limitations ranging from normal to over 15 degrees in flexion and over 10 degrees in rotation and extension were positive findings. The investigator used pinprick and soft touch to determine the presence of gross sensory deficit in the lower extremities.

Of the 229 patients selected, only 10 patients did not complete the treatment protocol. Reasons for noncompletion included transportation issues, family emergencies, scheduling conflicts, lack of motivation, and transient discomfort. The patient protocol provided for 20 treatments of spinal decompression over a 6-week course of therapy. Each session consisted of a 45-minute treatment on the equipment followed by 15 minutes of ice and interferential frequency therapy to consolidate the lumbar paravertebral muscles. The patient regimen included 2 weeks of daily spinal decompression treatment (5 days per week), followed by three sessions per week for 2 weeks, concluding with two sessions per week for the remaining 2 weeks of therapy.

On the first day of treatment, the applied pressure was measured as one half of the person´s body weight minus 10 pounds, followed on the second day with one half of the persons body weight. The pressure placed for the remainder of the 18 sessions was equivalent to one half of the patient´s body weight plus an additional 10 pounds. The angle of treatment was set according to manufacturer´s protocol after identifying a specific lumbar disc correlated with MRI findings. A session would begin with the patient being fitted with a customized lower and upper harness to fit their specific body frame. The patient would step onto a platform located at the base of the equipment, which simultaneously calculated body weight and determined proper treatment pressure. The patient was then lowered into the supine position, where the investigator would align the split of table with the top of the patient´s iliac crest. A pneumatic air pump was used to automatically increase lordosis of the lumbar spine for patient comfort. The patient´s chest harness was attached and tightened to the table. An automatic shoulder support system tightened and affixed the patient´s upper body. A knee pillow was placed to maintain slight flexion of the knees. With use of the previously calculated treatment pressures, spinal decompression was then applied. After treatment, the patient received 15 minutes of interferential frequency (80 to 120 Hz) therapy and cold packs to consolidate paravertebral muscles.

During the initial 2 weeks of treatment, the patients were instructed to wear lumbar support belts and limit activities, and were placed on light duty at work. In addition, they were prescribed a nonsteroidal, to be taken 1 hour before therapy and at bedtime during the first 2 weeks of treatment. After the second week of treatment, medication was decreased and moderate activity was permitted.

Data was collected from 219 patients treated during this clinical study. Study demographics consisted of 79 female and 140 male patients. The patients treated ranged from 24 to 74 years of age (see Table 1). The average weight of the females was 146 pounds and the average weight of the men was 195 pounds. According to the Oswestry Pain Scale, patients reported their symptoms ranging from no pain (0) to severe pain (5).

PATIENT GROUPS

The patients were further subdivided into six groups:

1. single lateral herniation 67 cases

2. single central herniation 22 cases

3. single lateral herniation
with disc degeneration 32 cases

4. single central herniation
with disc degeneration. 24 cases

5. more than 1 herniation
with disc degeneration 17 cases

6. more than 1 herniation
without disc degeneration 57 cases

RESULTS

According to the self-rated Oswestry Pain Scale, treatment was successful in 86% of the 219 patients included in this study (Table 2, page 39). Treatment success was defined by a reduction in pain to 0 or 1 on the pain scale. The perception of pain was none 0 to occasional 1 without any further need for medication or treatment in 188 patients. These patients reported complete resolution of pain, lumbar range of motion was normalized, and there was recovery of any sensory or motor loss. The remaining 31 patients reported significant pain and disability, despite some improvement in their overall pain and disability score.

Diagnosis MRI Findings	No. of CaseS	Female Patients	Male Patients	Positive Result	No Result	% of Success
Single Herniation Lateral	67	26	41	63	4	94
Single Herniation Central	22	11	11	20	2	90
Single Herniation w/ Degeneration	24	5	19	24	0	100
Single Herniation Lateral w/ Degeneration	32	14	18	29	3	91
Multiple Herniations w/o Degeneration	57	21	36	39	18	68
Multiple Herniations w/ Degeneration	17	2	15	13	4	77
TOTAL	219	79	140	188	31	86

Table 2. Results on self-rated Oswestry Pain Scale after treatment.

In this study, only patients diagnosed with herniated and degenerative discs with at least a 4-week onset were eligible. Each patient´s diagnosis was confirmed by MRI findings. All selected patients reported 3 to 5 on the pain scale with radiating neuritis into the lower extremities. By the second week of treatment, 77% of patients had a greater than 50% resolution of low back pain. Subsequent orthopedic examinations demonstrated that an increase in spinal range of motion directly correlated with an improvement in straight leg raises and reflex response. Table 2 shows a summary of the subjective findings obtained during this study by category and total results post treatment. After 90 days, only five patients (2%) were found to have relapsed from the initial treatment program.

Diagnosis MRI Findings	Improved Gait	Sluggish to Normal Reflexes	Improved Sensory Reception	Improved Motor Limitation	Abnormal to Normal Straight Leg Raise Test	Improved Spinal Range of Motion
Single Herniation Lateral	98%	98%	96%	90%	92%	95%
Single Herniation Central	100%	100%	94%	92%	96%	90%
Single Herniation w/ Degeneration	99%	96%	90%	84%	94%	90%
Single Herniation Lateral w/ Degeneration	94%	97%	94%	88%	90%	92%
Multiple Herniations w/o Degeneration	96%	94%	94%	81%	82%	92%
Multiple Herniations w/ Degeneration	92%	94%	88%	82%	80%	82%
AVERAGE IMPROVEMENT	96%	96%	93%	86%	89%	90%

Table 3. Percentage of patients that had improved physical exam findings post treatment.

Ninety-two percent of patients with abnormal physical findings improved post-treatment. Ninety days later only 3% of these patients had abnormal findings. Table 3 summarizes the percentage of patients that showed improvement in physician examination findings testing both motor and sensory system function after treatment. Gait improved in 96% of the individuals who started with an abnormal gait, while 96% of those with sluggish reflexes normalized. Sensory perception improved in 93% of the patients, motor limitation diminished in 86%, 89% had a normal straight leg raise test who initially tested abnormal, and 90% showed improvement in their spinal range of motion.

FREQUENTLY ASKED QUESTIONS

What is the difference between decompression and traction?

Many clinicians specializing in lumbar spine pathology have criticized traditional traction. Traction fails in many cases because it causes muscular stretch receptors to fire, which then cause para-spinal muscles to contract. This muscular response actually causes an increase in intradiscal pressure. On the other hand, genuine decompression is achieved by gradual and calculated increases of distraction forces to spinal structures, utilizing various degrees of distraction forces.

A highly specialized computer must modulate the application of distraction forces in order to achieve the ideal effect. The system uses applies a gentle, curved angle pull which yields far greater treatment results that a less comfortable, sharp angle pull. Distraction must be offset by cycles of partial relaxation.

The system continuously monitors spinal resistance and adjusts distraction forces accordingly. A specific lumbar segment can be targeted for treatment by changing the angle of distraction. This patented technique of decompression may prevent muscle spasm and patient guarding. Constant activity monitoring takes place at a rate of 10,000 times per second, making adjustments not perceived by the eye as many as 20 times per second via its fractional metering and monitoring system.

Genuine decompression also involves the use of a special pelvic harness that supports the lumbar spine during therapy. Negative pressure within the disc is maintained throughout the treatment session. With genuine decompression, the pressure within the disc space can actually be lowered to about -150 mmHg. As a result, the damaged disc will be rehydrated with nutrients and oxygen.

Isn´t decompression just a fancy name for a traction machine?

No. There is a big different between traction, distraction and decompression. Traction has been around for hundreds, if not thousands of years. The problem with traction as it is known today is that it is not always beneficial. In 1998, the Scientific American rated traction to be of little or no value in the examination of efficacious therapies for lower back pain. This finding is consistent with many studies that report traction can often times signal a nociceptive splinting response and put a patient’s back muscles in spasm, resisting any attempts to effect a change on the disc proper.

Distraction, a term used to describe a flexion distraction technique, attempts to reposition the spine from the offending lesion. This technique has been shown to be very effective, even though potentially damaging to the person performing the technique and largely dependent on the skill of the technician. Like traction, distraction procedures are limited in the ability to reduce the intradiscal pressure, or produce a negative pressure within the disc imbibing fluid, nutrients and creating an environment for repair.

Decompression therefore is an event - a combination of restraint, angle position and equipment engineering. One can experience traction without decompression, but not decompression without traction. Traction is a machine - Decompression is an event.

What Result can I Expect?

Many patients with lower back syndromes may experience pain relief as early as the third treatment session. Comparison of pre-treatment MRI´s with post-treatment MRI´s has shown a 50% reduction in the size and extent of herniation. In clinical studies, 86% of patients reported relief of back pain with the our system. Within the past five years, some private practice clinicians have reported success rates as high as 90%.

What Time Commitments Are Required By Patients?

Each treatment session averages 25 to 30 minutes in duration (research has established that optimum results are achieved with sessions that incorporate 10 to 15 decompression/relaxation cycles). On average, one daily session for 20-30 treatments is necessary for patient self-healing to occur.

Herniated discs generally respond within 20 sessions, while patients with degenerated discs may need ongoing therapy at regulated intervals to remain pain free. Still other patients, due to lifestyle or occupation, may also require maintenance therapy. Patients with posterior facet syndromes may achieve complete remission with 10 or fewer sessions. Research has demonstrated that most patients achieve full remission from pain after the initial treatment regimen.

What is the typical diagnosis?

Since non-specific low back pain and cervical pain generally encompass a myriad of mechanical failures, including muscles, tendons, ligaments, and other soft tissue that encroach or produce pressure on the nerves, the term intervertebral disc syndrome can be used. This diagnosis does not necessarily require (although recommended) an MRI to confirm the presence of a disc involvement

Who can benefit from using Disc Decompression Therapy?

The following would be inclusion criteria for the Decompression Therapy (1) Pain due to herniated and bulging lumbar discs that is more than four weeks old; (2) Recurrent pain from a failed back surgery that is more than six months old; (3) Persistent pain from degenerated discs not responding to four weeks of therapy; (4) Patients available for four weeks of treatment protocol; and (5) Patient at least 18 years of age.

These indications are ideal candidates for enrollment into our program and have the potential of achieving quality outcomes in the treatment of their back pain: (1) Nerve Compression; (2) Lumbar Disorders; (3) Lumbar Strains; (4) Sciatic Neuralgia; (5) Herniated Discs; (6) Injury of the Lumbar Nerve Root; (7) Degenerative Discs; (8) Spinal Arthritis; (9) Low Back Pain w/ or w/o Sciatica; (10) Degenerative Joint Disease; (11) Myofasctois Syndrome; (12) Disuse Atrophy; (13) Lumbar Instability; (14) Acute Low Back Pain; and (15) Post-Surgical Low Back Pain.

Lastly, the system should be utilized with patients with low back pain, with or without radiculopathy who have failed conventional therapy (physiotherapy and chiropractic) and who are considering surgery. Surgery should only be considered following a reasonable trial of Decompression therapy protocols.

What conditions are contraindicated?

Patients with the following problems or symptoms are usually excluded from using the Spinal Decompresion therapy: Pregnancy, Prior lumbar surgical fusion, Metastatic cancer, Severe osteoporosis, Compression fracture of lumbar spine below L-1, Pars defect, Aortic aneurysm, Pelvic or abdominal cancer, Disc space infections, Severe peripheral neuropathy, Hemiplegia, paraplegia, or cognitive dysfunction, Cauda Equina syndrome, Tumors, osteod osteoma, multiple myeloma, osteosarcoma, Infection, osteomyelitis, meningitis, virus, and HNP (sequestered/free floating fragment).

How long is each session and what is the treatment protocol?

Each session on the Decompression equipment is approximately 25-40 minutes long (45 minute sessions include set-up and take-off), accompanied by 15 minutes of stimulation, heat packs and manipulation. The patient comes for 20-30 visits over a 4-6 week period. The doctor will provide a complete copy of the Spinal decompression treatment protocol upon request.

How long before a patient experiences change?

Often times a patient experiences some relief within the first few (3-7) treatments. Usually by the 12th to 15th treatment all patients have reported some remission of symptoms. Patients not showing significant improvement by the 15th to 18th session may be referred for further diagnostic evaluation.

Does Decompression Therapy work for everyone?

Eighty-to-ninety percent of patients who have been properly selected and comply with the Spinal Disc Decompression protocol will have good-to-excellent outcomes. Patient´s conditions that do not respond quickly to the therapy are often unable to be helped by anything quickly. Patients vary in age, sex and body morphology and may require counseling in weight loss, nutrition and other lifestyle changes.

Regain your good health and get your life BACK! 

Disc herniation signs and symptoms

If you sneeze, Cough or bear down and it hurts this is usually an indication that the disc is involved. Patients can have Back pain with or without leg pain and leg pain and weakness without  back pain.

Why doesn't insurance pay for Spinal Decompression?

As with most new procedures like Lasik eye surgery or gastric bypass the insurance companies take there time in reimbursing for these procedures. We all know that Lasik eye surgery works but most insurance still do not pay for it. Some carriers are starting to pay for Spinal Decompression.

Do I need a MRI?

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Although a recent MRI has become the gold standard to document presence of a herniated disc, their are abnormal types of movements that can be shown on radiographs while the patient is holding their spine in full extension and flexion.  The two radiologic change that are indicative of instability, vacum sign (Knuttson's phemenom of gas in the disc) and the "traction spur" also known as the Macnab spur. So if a recent MRI is not available the use of a radiograph can point to a lumbar instability. I recently purchased a high speed low dose digital system for use in my practice.